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Name: DR:AIJAZ Gender/age: M/22 City/Country: HYDERABAD/PAKISTAN Year of Graduation/institute: 2004 Present position: WORKING AS DOCTOR IN AGHA KHAN UNIV HOSP Specialty: FCPS-I, CLEARED NOW COPM;ET REQURIMENTS Qualifications: MBBS+FCPS-I E-mail address: z_chachar@hotmail.com Phone No (Off & RES): 0221-656633 Any other information:
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Osteogenesis Imperfecta (OI) or brittle bone disease is a group of autosomal dominant inherited disorders with abnormality in Type 1 collagen.[1], which usually result in bone fragility[2]. Emedicine Frequency of OI in USA and worldwide, clinical features, laboratory diagnosis, classification and differential diagnosis. The Cleveland Clinic Explains the types and management of the Osteogenesis Imperfecta… Read More »
According to Australian Government Department of Health and Ageing (2001). The main reason people were dropping private health insurance is the raising cost of premiums. The introduction of rebate will make private health insurance more easily affordable to both new and existing members and will halt the decline in the participation rate. It will give… Read More »
Health expenditure According to Harper (2003) health is a large component of the Australian economy currently involving well over $40 billion of public and private expenditure (or 8.5% of GDP), around 32% of which is privately funded. The cost to Government of the rebate must be seen in this context and reviewed against the… Read More »
Private health insurance in Australia and New Zealand. According to Quicken (2003) there will be: Less waiting time for elective surgeries. Facilities like a TV or phone in hospitals can be provided. People can afford choose the doctor of there choice. Patients can be treated in a private ward. Some funds also cover you… Read More »
Definition : Rheumatoid arthritis (RA) is a chronic systemic inflammatory disease of undetermined aetiology involving primarily the synovial membranes and articular structures of multiple joints. The disease is often progressive and results in pain, stiffness, and swelling of joints. In late stages deformity and ankylosis develop. Incidence and Prevalence: Worldwide prevalence is approx 1%. Its… Read More »
Clinical manifestation: There are two clinical phases which characterizes PWS as shown in table 1 (Zellweger, 1988): The two clinical phases of PWS (1) Phase 1 Moderate to severe neonatal hypotonia Poor suck Difficulty in feeding Hypogonadism Marked delayed in motor milestone Phase 2 Much less hypotonia Short stature Hyperphagia Increasing obesity… Read More »
PWS is a complex disorder with variable phenotypic changes according to the age of the patient. There are numbers of Associations and Support groups are working together to give a better management to the PWS individuals e.g. Prader Willi Syndrome Association (UK) and PWSA (USA) etc. Researches are still in progress in all concerned… Read More »
Care providers of a PWS patient must understand that the intervention means the management of clinical symptoms. Because PWS undergoes different developmental changes they must understand its impact on both patient and their families. Creation of a management strategy should be drown on the condition of disease. Professional experiences and from the knowledge of… Read More »
Over past decade it has been noticed that structural and functional homologies extend across very distantly related species. Now day’s homology to a relevant orthologus gene is a criterion of extracting the candidency of a gene to a disease. Virtually every mouse gene has the exact counterparts as human, and the same probably do… Read More »
The etiology and pathology of disease is not well understood until very recent advances in the field of cytogenetics and molecular genetics. These recent advances are now providing some insight of the fairly complicated syndrome. Approximately 70% of the PWS have been shown to have a deletion in chromosome 15, described as 15q11-q13.… Read More »
(1) This case study has been provided by Jack Sherman and adapted from Greenswag & Alexander, 1995. In 1957, just after the publication of the article by Prader, Labhart and Willi (1956) Miss M. was born. Due to lack of information and awareness it took a long time to be diagnosed. M. was evaluated… Read More »
Many of the child behavior problems and cognitive capabilities can be explained within the framework of understanding the characteristic of Prader Willi Syndrome. Society, family and hereditary factors also influence it. PWS is a disease of altered growth and development. Affected persons are potentially overweight, sexually immature, and short and developmentally delayed individuals. The… Read More »
Table of contents: Introduction: Nature of Prader Willi Syndrome: Etiology and Pathogenesis: Genetics of Prader Willi Syndrome: Animal Model: Diagnostic Criteria: Case Study: Management: Conclusion:
1. Fader, Henry C., Computerized patient records. [online] URL http://www.physiciansnews.com/computers/1197.html [Accessed On Web 13 October 2004 ]. 2. Ginneken van, Astrid M., 2002. The computerized patient record: balancing effort and benefit. International Journal of Medical Informatics [online], 65 (2002). Available from: http://www.sciencedirect.com/science?_ob=Mimg&_imagekey=B6T7S/ [Accessed 13 October 2004 ]. 3. Hilton, David, Information… Read More »